Human Pathology
Volume 41, Issue 1 , Pages 134-138 , January 2010

Prominent Mott cell proliferation in Epstein-Barr virus-associated gastric carcinoma

Received 13 April 2009 ,Revised 3 July 2009 ,Accepted 9 July 2009.

References 

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  2. Fukayama M, Hino R, Uozaki H. Epstein-Barr virus and gastric carcinoma: virus-host interactions leading to carcinoma. Cancer Sci. 2008;99:1726–1733
  3. Ohtani H, Jin Z, Takegawa S, Nakayama T, Yoshie O. Abundant expression of CXCL9 (MIG) by stromal cells that include dendritic cells and accumulation of CXCR3+ T cells in lymphocyte-rich gastric carcinoma. J Pathol. 2009;217:21–31
  4. Nakamura S, Aoyagi K, Iwanaga S, Yao T, Tsuneyoshi M, Fujishima M. Synchronous and metachronous primary gastric lymphoma and adenocarcinoma: a clinicopathological study of 12 patients. Cancer. 1997;79:1077–1085
  5. Tazawa K, Tsutsumi Y. Localized accumulation of Russell body-containing plasma cells in gastric mucosa with Helicobacter pylori infection: “Russell body gastritis.”. Pathol Int. 1998;48:242–244
  6. Paik S, Kim SH, Kim JH, Yang WI, Lee YC. Russell body gastritis associated with Helicobacter pylori infection: a case report. J Clin Pathol. 2006;59:1316–1319
  7. Pizzolitto S, Camilot D, DeMaglio G, Falconieri G. Russell body gastritis: expanding the spectrum of Helicobacter pylori–related diseases?. Pathol Res Pract. 2007;203:457–460
  8. Willems S, Carneiro F, Geboes K. Gastric carcinoma with osteoclast-like giant cells and lymphoepithelioma-like carcinoma of the stomach: two of a kind?. Histopathology. 2005;47:331–333

PII: S0046-8177(09)00253-6

doi: 10.1016/j.humpath.2009.07.004

Human Pathology
Volume 41, Issue 1 , Pages 134-138 , January 2010

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